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authorBonface2024-02-13 23:52:26 -0600
committerMunyoki Kilyungi2024-08-09 13:30:43 +0300
commitb2feda451ccfbeaed02dce9088d6dd228cf15861 (patch)
tree3dd2883524985114070a7770cd2e9f9bd7eb1848 /general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf
parentd029d5d7f8ead1f1de8d318045004a4a6f68f5fb (diff)
downloadgn-docs-b2feda451ccfbeaed02dce9088d6dd228cf15861.tar.gz
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+<p>Glaucoma is characterized by the progressive dysfunction and loss of retinal ganglion cells. Recent work in animal models suggests that a critical neuroinflammatory event damages retinal ganglion cell axons in the optic nerve head during ocular hypertensive injury. We previously demonstrated that monocyte-like cells enter the optic nerve head in an ocular hypertensive mouse model of glaucoma (DBA/2&thinsp;J), but their roles, if any, in mediating axon damage remain unclear.</p>
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+<p><a href="https://molecularneurodegeneration.biomedcentral.com/articles/10.1186/s13024-018-0303-3" target="_blank">https://molecularneurodegeneration.biomedcentral.com/articles/10.1186/s13024-018-0303-3</a></p>