From b2feda451ccfbeaed02dce9088d6dd228cf15861 Mon Sep 17 00:00:00 2001
From: Bonface
Date: Tue, 13 Feb 2024 23:52:26 -0600
Subject: Update dataset RTF Files.
---
general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf | 3 +++
1 file changed, 3 insertions(+)
create mode 100644 general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf
(limited to 'general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf')
diff --git a/general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf b/general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf
new file mode 100644
index 0000000..b5e090d
--- /dev/null
+++ b/general/datasets/Jax_d2_mono1vpbmc_ret_rna_seq_0619/summary.rtf
@@ -0,0 +1,3 @@
+Glaucoma is characterized by the progressive dysfunction and loss of retinal ganglion cells. Recent work in animal models suggests that a critical neuroinflammatory event damages retinal ganglion cell axons in the optic nerve head during ocular hypertensive injury. We previously demonstrated that monocyte-like cells enter the optic nerve head in an ocular hypertensive mouse model of glaucoma (DBA/2 J), but their roles, if any, in mediating axon damage remain unclear.
+
+https://molecularneurodegeneration.biomedcentral.com/articles/10.1186/s13024-018-0303-3
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