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height:35.23%'><span style='font-size:68%'><i>Is there known biology to link
Hars2 with App? </i></span></div>
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<div style='position:absolute;top:0%;left:0%;width:100.0%;height:34.28%'><span
style='font-size:150%;color:#E9EB5D;mso-color-index:3'><b>69 SNPs, 1 cSNP:
<br>
</b></span></div>
<div style='position:absolute;top:32.85%;left:0%;width:96.81%;height:34.28%'><span
style='font-size:150%;color:#E9EB5D;mso-color-index:3'><b>6 exons in NCBI,
<br>
</b></span></div>
<div style='position:absolute;top:65.71%;left:0%;width:100.0%;height:34.28%'><span
style='font-size:150%;color:#E9EB5D;mso-color-index:3'><b>2 exons in Celera</b></span></div>
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height:6.53%'><span style='font-family:"Gill Sans";font-size:250%;color:#E9EB5D'><i><span
style="mso-spacerun: yes"> </span>Not obvious</i></span></div>
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<td align=left colspan=1><font face=Helvetica size=3>Hars2 is not a well
characterized gene and their is no biology yet to support the hypothesis that
Hars2 modulates gene expression, let alone App expression in specific. There
are also serious database/assembly discrepancies between Celera and MGSCv3
regarding the genomic organization of this gene. But there appear to be
approximately 69 SNPs in Hars2, one of which results in a substitution.</font><br>
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